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Institute of Physiology and Pathophysiology

Prof. Dr. med. Oliver Kann

Heidelberg University

Institute of Physiologie and Pathophysiology

WG General Neurophysiology

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

phone:

+49 6221 54-4560

fax:

+49 6221 54-6364

e-mail:

oliver.kann@physiologie.uni-heidelberg.de

since 2010

Full Professor for General Neurophysiology (W3), Medical Faculty Heidelberg, Ruprecht-Karls-University, Germany

2009

Habilitation and venia legendi for Physiology, Charité-Universitätsmedizin Berlin, Germany

2005-2006

Guest scientist, Department of Neurobiology, Koret School of Veterinary Medicine, The Hebrew University of Jerusalem, Rehovot, Israel 

2004-2010

Senior Research Assistent, Neuronal Mitochondria Research Group (PI), Institute for Neurophysiology, Charité-Universitätsmedizin Berlin 

2003

Licence to practise medicine

2001

Conferral of doctorate (MD), Humboldt-University of Berlin 

2000-2004

Research Assistent, Institute for Neurophysiology and Department of  Neurology, Charité, Humboldt-University of Berlin, Germany

2000

State examination (Staatsexamen) in Human Medicine 

1999

Medical internship (internal medicine, surgery and neurology)

1998

Intermediate examination (Zwischenprüfung) in Philosophy 

1996-1998

Experimental work for dissertation, Department of Cellular Neurosciences, Max-Delbrück-Center for Molecular Medicine, Berlin-Buch, Germany

1992-2000

Studies of Human Medicine and Philosophy, Philipps-University of Marburg and Free-University of Berlin, Germany

Honors and Awards

2010

Teaching award of reformed curriculum, Charité-Universitätsmedizin Berlin 

2008

Poster award, Gordon Research Conference (Andover, USA) 

2006

Poster award, Gordon Research Conference (Oxford, UK) 

2005

Minerva Short-Term Research Grant (Max-Planck-Society)

1996

Scholarship by GRK-238 (German Research Foundation)


Recent Publications

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Methylglyoxal evokes acute Ca2+ transients in distinct cell types and increases agonist-evoked Ca2+ entry in endothelial cells via CRAC channels. Cell Calcium. 2019 Mar;78:66-75. doi: 10.1016/j.ceca.2019.01.002. Epub 2019 Jan 9.

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EphB2-dependent signaling promotes neuronal excitotoxicity and inflammation in the acute phase of ischemic stroke. Acta Neuropathol Commun. 2019 Feb 5;7(1):15. doi: 10.1186/s40478-019-0669-7.

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Early alterations in hippocampal perisomatic GABAergic synapses and network oscillations in a mouse model of Alzheimer's disease amyloidosis. PLoS One. 2019 Jan 15;14(1):e0209228. doi: 10.1371/journal.pone.0209228. eCollection 2019.

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Medikamentöse Varikosetherapie aus der Perspektive experimenteller Modelle. Praxis (Bern 1994). 2019 Jan;108(1):31-36. doi: 10.1024/1661-8157/a003147.

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Selective vulnerability of αOFF retinal ganglion cells during onset of autoimmune optic neuritis. Neuroscience. 2018 Nov 21;393:258-272. doi: 10.1016/j.neuroscience.2018.07.040. Epub 2018 Aug 1.

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The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function. FASEB J2018 Nov;32(11):6159-6173. Epub 2018 Jun 7.

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Genetic ablation of NFAT5/TonEBP in smooth muscle cells impairs flow- and pressure-induced arterial remodeling in mice. FASEB J. 2018 Nov 1:fj201801594R. doi: 10.1096/fj.201801594R. [Epub ahead of print]

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Reduction of Transplant Vasculopathy by Intraoperative Nucleic Acid-based Therapy in a Mouse Aortic Allograft Model. Thorac Cardiovasc Surg. 2018 Oct 23. doi: 10.1055/s-0038-1673633. [Epub ahead of print]


Institute of
Physiology and Pathophysiology

Heidelberg University

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

Phone:+49 6221 54-4035
Fax:+49 6221 54-4038
E-mail:sekretariat.hecker@
physiologie.uni-heidelberg.de