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Institute of Physiology and Pathophysiology

Prof. Dr. med. Oliver Kann

Heidelberg University

Institute of Physiologie and Pathophysiology

WG General Neurophysiology

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

phone:

+49 6221 54-4560

fax:

+49 6221 54-6364

e-mail:

oliver.kann@physiologie.uni-heidelberg.de

since 2010

Full Professor for General Neurophysiology (W3), Medical Faculty Heidelberg, Ruprecht-Karls-University, Germany

2009

Habilitation and venia legendi for Physiology, Charité-Universitätsmedizin Berlin, Germany

2005-2006

Guest scientist, Department of Neurobiology, Koret School of Veterinary Medicine, The Hebrew University of Jerusalem, Rehovot, Israel 

2004-2010

Senior Research Assistent, Neuronal Mitochondria Research Group (PI), Institute for Neurophysiology, Charité-Universitätsmedizin Berlin 

2003

Licence to practise medicine

2001

Conferral of doctorate (MD), Humboldt-University of Berlin 

2000-2004

Research Assistent, Institute for Neurophysiology and Department of  Neurology, Charité, Humboldt-University of Berlin, Germany

2000

State examination (Staatsexamen) in Human Medicine 

1999

Medical internship (internal medicine, surgery and neurology)

1998

Intermediate examination (Zwischenprüfung) in Philosophy 

1996-1998

Experimental work for dissertation, Department of Cellular Neurosciences, Max-Delbrück-Center for Molecular Medicine, Berlin-Buch, Germany

1992-2000

Studies of Human Medicine and Philosophy, Philipps-University of Marburg and Free-University of Berlin, Germany

Honors and Awards

2010

Teaching award of reformed curriculum, Charité-Universitätsmedizin Berlin 

2008

Poster award, Gordon Research Conference (Andover, USA) 

2006

Poster award, Gordon Research Conference (Oxford, UK) 

2005

Minerva Short-Term Research Grant (Max-Planck-Society)

1996

Scholarship by GRK-238 (German Research Foundation)


Recent Publications

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Metabolic modulation of neuronal gamma-band oscillations. Pflugers Arch2018 Sep;470(9):1377-1389. doi: 10.1007/s00424-018-2156-6. Epub 2018 May 28.

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Electrical coupling between hippocampal neurons: contrasting roles of principal cell gap junctions and interneuron gap junctions. Cell Tissue Res. 2018 Aug 15. doi: 10.1007/s00441-018-2881-3. [Epub ahead of print] Review.

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Selective vulnerability of αOFF retinal ganglion cells during onset of autoimmune optic neuritis. Neuroscience. 2018 Jul 31. pii: S0306-4522(18)30515-3. doi: 10.1016/j.neuroscience.2018.07.040. [Epub ahead of print]

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Strategy for marker-based differentiation of pro- and anti-inflammatory macrophages using matrix-assisted laser desorption/ionization mass spectrometry imaging. Analyst. 2018 Jul 20. doi: 10.1039/c8an00659h. [Epub ahead of print]

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Improving electrical properties of iPSC-cardiomyocytes by enhancing Cx43 expression. J Mol Cell Cardiol. 2018 Jul;120:31-41. doi: 10.1016/j.yjmcc.2018.05.010. Epub 2018 May 16.

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Role of CD40 and ADAMTS13 in von Willebrand factor-mediated endothelial cell-platelet-monocyte interaction. Proc Natl Acad Sci U S A. 2018 Jun 12;115(24):E5556-E5565. doi: 10.1073/pnas.1801366115. Epub 2018 May 23.

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The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function. FASEB J. 2018 Jun 7:fj201800246R. doi: 10.1096/fj.201800246R. [Epub ahead of print]

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Persistent sodium current modulates axonal excitability in CA1 pyramidal neurons. J Neurochem. 2018 Jun 4. doi: 10.1111/jnc.14479. [Epub ahead of print]

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The lncRNA CASC9 and RNA binding protein HNRNPL form a complex and co-regulate genes linked to AKT signaling. Hepatology. 2018 May 23. doi: 10.1002/hep.30102. [Epub ahead of print]

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Early Blood-Brain Barrier Disruption in Ischemic Stroke Initiates Multifocally Around Capillaries/Venules. Stroke. 2018 Jun;49(6):1479-1487. doi: 10.1161/STROKEAHA.118.020927. Epub 2018 May 14.

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Impact of carbonylation on glutathione peroxidase-1 activity in human hyperglycemic endothelial cells. Redox Biol. 2018 Jun;16:113-122. doi: 10.1016/j.redox.2018.02.018. Epub 2018 Mar 1.

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Endothelial progenitor cells accelerate endothelial regeneration in an in vitro model of Shigatoxin-2a-induced injury via soluble growth factors. Am J Physiol Renal Physiol. 2018 Mar 7. doi: 10.1152/ajprenal.00633.2017. [Epub ahead of print]


Institute of
Physiology and Pathophysiology

Heidelberg University

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

Phone:+49 6221 54-4035
Fax:+49 6221 54-4038
E-mail:sekretariat.hecker@
physiologie.uni-heidelberg.de