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Institute of Physiology and Pathophysiology

Institute of Physiology and Pathophysiology

Heidelberg University

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

 

 

Managing Director

 

Prof. Dr. Markus Hecker

 

phone:

+49 6221 54-4036

fax:

+49 6221 54-4038

e-mail:

hecker@physiologie.uni-heidelberg.de



Secretary

 

Barbara Richards

 

phone:

+49 6221 54-4035

fax:

+49 6221 54-4038

e-mail:

sekretariat.hecker@physiologie.uni-heidelberg.de

The Institute for Physiology and Pathophysiology is a scientific institution assigned to the Faculty of Medicine of Heidelberg University. Its tasks are education and research in physiology.

 

About 100 people work in two divisions, headed by the Professors Dr. Andreas Draguhn and Dr. Markus Hecker, their associated independent working groups lead by the Professors Dr. Oliver Kann, Dr. Alexander Groh and Dr. Hugo Marti, respectively, and the administration.

 

Members of the institute jointly teach lecture and lab courses in physiology for the approximately 450 students of medicine and dentistry admitted annually to Heidelberg University.

 

 

The short animated video "What is physiology" produced by The Physiological Society (UK) gives a brief introduction into the field of physiology. To watch the video on the society's YouTube channel click here.Dies ist ein externer Link

by kind permission of The Physiological Society (UK)Dies ist ein externer Link


Recent Publications

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Chronic hypoxia changes gene expression profile of primary rat carotid body cells: consequences on the expression of NOS isoforms and ET-1 receptors. Physiol Genomics. 2019 Mar 1. doi: 10.1152/physiolgenomics.00114.2018. [Epub ahead of print]

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Priming of microglia with IFN-γ slows neuronal gamma oscillations in situ. Proc Natl Acad Sci U S A. 2019 Feb 19. pii: 201813562. doi: 10.1073/pnas.1813562116. [Epub ahead of print]

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Methylglyoxal evokes acute Ca2+ transients in distinct cell types and increases agonist-evoked Ca2+ entry in endothelial cells via CRAC channels. Cell Calcium. 2019 Mar;78:66-75. doi: 10.1016/j.ceca.2019.01.002. Epub 2019 Jan 9.

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EphB2-dependent signaling promotes neuronal excitotoxicity and inflammation in the acute phase of ischemic stroke. Acta Neuropathol Commun. 2019 Feb 5;7(1):15. doi: 10.1186/s40478-019-0669-7.

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Early alterations in hippocampal perisomatic GABAergic synapses and network oscillations in a mouse model of Alzheimer's disease amyloidosis. PLoS One. 2019 Jan 15;14(1):e0209228. doi: 10.1371/journal.pone.0209228. eCollection 2019.

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Medikamentöse Varikosetherapie aus der Perspektive experimenteller Modelle. Praxis (Bern 1994). 2019 Jan;108(1):31-36. doi: 10.1024/1661-8157/a003147.

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Selective vulnerability of αOFF retinal ganglion cells during onset of autoimmune optic neuritis. Neuroscience. 2018 Nov 21;393:258-272. doi: 10.1016/j.neuroscience.2018.07.040. Epub 2018 Aug 1.

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The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function. FASEB J2018 Nov;32(11):6159-6173. Epub 2018 Jun 7.

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Genetic ablation of NFAT5/TonEBP in smooth muscle cells impairs flow- and pressure-induced arterial remodeling in mice. FASEB J. 2018 Nov 1:fj201801594R. doi: 10.1096/fj.201801594R. [Epub ahead of print]

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Reduction of Transplant Vasculopathy by Intraoperative Nucleic Acid-based Therapy in a Mouse Aortic Allograft Model. Thorac Cardiovasc Surg. 2018 Oct 23. doi: 10.1055/s-0038-1673633. [Epub ahead of print]


Institute of
Physiology and Pathophysiology

Heidelberg University

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

Phone:+49 6221 54-4035
Fax:+49 6221 54-4038
E-mail:sekretariat.hecker@
physiologie.uni-heidelberg.de