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Institute of Physiology and Pathophysiology

Prof. Dr. med. Andreas Draguhn

Heidelberg University

Institute of Physiology and Pathophysiology

Division of Neuro- and Sensory Physiology

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

phone:

+49 6221 54-4057/4056

Telefax:

+49 6221 54-6364

E-Mail:

andreas.draguhn@physiologie.uni-heidelberg.de

since 2016

Managing Director of the Institute of Physiology and Pathophysiology, Heidelberg University, Germany

2012-2013

Managing Director of the Institute of Physiology and Pathophysiology, Heidelberg University, Germany

since 2002

Full professor and Director at the Institute of Physiology and Pathophysiology, Heidelberg University, Germany

2001

Offer for full professorship at the Free University of Berlin, Germany (declined)

1999

State Doctorate (Habilitation)

1997

Visiting scientist at Birmingham University, UK (Prof. John G. R. Jefferys)

1994-1997

Assistant and group leader, Institute of Physiology at the Charité, Berlin, Germany

1993

Full medical license

1992-1994

Postdoctoral Fellow, Institute of Physiology, Cologne University, Germany

1992

MD “Summa cum laude“ at the Medical Faculty of Heidelberg University

1991

Preliminary approbation as MD

1987-1990

Dissertation in Göttingen and Heidelberg, Germany

1984–1986

Student assistent at the Institute of Physiology, Bonn University, Germany

1981-1987

Studies in Medicine, Physics and Philosophy at Bonn University, Germany


Recent Publications

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Methylglyoxal evokes acute Ca2+ transients in distinct cell types and increases agonist-evoked Ca2+ entry in endothelial cells via CRAC channels. Cell Calcium. 2019 Mar;78:66-75. doi: 10.1016/j.ceca.2019.01.002. Epub 2019 Jan 9.

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EphB2-dependent signaling promotes neuronal excitotoxicity and inflammation in the acute phase of ischemic stroke. Acta Neuropathol Commun. 2019 Feb 5;7(1):15. doi: 10.1186/s40478-019-0669-7.

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Early alterations in hippocampal perisomatic GABAergic synapses and network oscillations in a mouse model of Alzheimer's disease amyloidosis. PLoS One. 2019 Jan 15;14(1):e0209228. doi: 10.1371/journal.pone.0209228. eCollection 2019.

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Medikamentöse Varikosetherapie aus der Perspektive experimenteller Modelle. Praxis (Bern 1994). 2019 Jan;108(1):31-36. doi: 10.1024/1661-8157/a003147.

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Selective vulnerability of αOFF retinal ganglion cells during onset of autoimmune optic neuritis. Neuroscience. 2018 Nov 21;393:258-272. doi: 10.1016/j.neuroscience.2018.07.040. Epub 2018 Aug 1.

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The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function. FASEB J2018 Nov;32(11):6159-6173. Epub 2018 Jun 7.

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Genetic ablation of NFAT5/TonEBP in smooth muscle cells impairs flow- and pressure-induced arterial remodeling in mice. FASEB J. 2018 Nov 1:fj201801594R. doi: 10.1096/fj.201801594R. [Epub ahead of print]

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Reduction of Transplant Vasculopathy by Intraoperative Nucleic Acid-based Therapy in a Mouse Aortic Allograft Model. Thorac Cardiovasc Surg. 2018 Oct 23. doi: 10.1055/s-0038-1673633. [Epub ahead of print]


Institute of
Physiology and Pathophysiology

Heidelberg University

Im Neuenheimer Feld 326

69120 Heidelberg

Germany

Phone:+49 6221 54-4035
Fax:+49 6221 54-4038
E-mail:sekretariat.hecker@
physiologie.uni-heidelberg.de